Bone Marrow Defects Seen in Rheumatoid Arthritis, Might Hinder Experimental Therapies

NEW YORK (Reuters Health) Mar 22 – Patients with active rheumatoid arthritis have quantitative and qualitative abnormalities of bone marrow hematopoietic progenitor cells, and the bone marrow microenvironment is unable to support normal hematopoiesis, study results show.
The abnormalities are due, at least in part, to increased production of tumor necrosis factor alpha (TNF-alpha) by inflammatory cells within the bone marrow, according to Dr. Helen A. Papadaki, of University Hospital of Heraklion, Greece, and colleagues there and at St. George's Hospital Medical School in London.

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